The HIV variant might be more contagious, but it responds to treatment like similar strains and is not necessarily more deadly.
Through genetic sequencing, the researchers surmised that the variant arose from a series of mutations over time, Wymant says. However, the researchers weren’t able to identify which specific mutations occurred in subtype B to create this new variant. This new variant is very different from all the other variants of subtype B, according to Wymant.
“Because all of those mutations have happened pretty much together, we don’t know which of them is the one that’s responsible for [its virulence],” he says. Wymant hopes that future research can identify which specific mutations occurred to make this variant is so severe. The rate of CD4“So that might be because there’s something slightly different happening at a molecular or cellular level,” he says.
That said, Wymant emphasizes that this discovery, like COVID-19 variants, shows how important it is to stop the spread of a pathogen before it mutates. “Viruses and bacteria can surprise us with their evolution. They can turn into something which is worse for us,” Wymant says. “Every time you prevent a pathogen from passing from one person to the next, you’re not only saving that person from the infection that they would have had, but you’re denying that pathogen an opportunity to evolve into something else.”
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